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The rhythmic acetylation of BMAL1 by CLOCK supports recruitment of the CRY1 protein to the CLOCK/BMAL1 heterodimer complex, promoting transcriptional silencing of CLOCK target genes, as has been shown in the liver (Hirayama et al., Per1 gene by bringing the transcriptional co-repressor SIN3 transcription regulator family member B (SIN3B) and HDAC1/2 to its promoter (Naruse et al., Per1 promoter is also regulated by SIN3A and HDAC1, which are recruited by period circadian clock 2 (Per2) and polypyrimidine tract-binding protein-associated splicing factor (PSF
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