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Recently, Volterra’s group (de Ceglia et al., 2023) demonstrated in the hippocampus the existence of a subset of astrocytes that performs exocytotic release of glutamate following astrocyte-selective stimulations
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The hypothesis that these reactive intermediates are responsible for cell death is supported by the observation that a cell line selected for erastin resistance showed several-hundred-fold upregulation of AKR1C family genes (Dixon et al., 2014), which encode aldoketoreductases that, among other functions, reduce reactive end-products of lipid peroxidation to unreactive compounds (MacLeod et al., 2009)
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