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Valproic acid and other histone deacetylase inhibitors induce microglial apoptosis and attenuate lipopolysaccharide-induced dopaminergic neurotoxicity
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These mechanisms suggest that although exercise and fluoxetine act through different means, they share multiple molecular pathways centered on the 5-HT system, exerting additive or complementary effects on key processes such as Aβ metabolism, tau pathology, neuroinflammation, neurotrophic support and cholinergic function, thus providing a systematic theoretical foundation for combined interventions in AD
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